Pathogenesis of Influenza Virus

Influenza infection spreads from a contaminated individual, it ties to and reproduces in epithelial cells of both the upper and lower respiratory tract. Viral replication consolidated with the resistant reaction to disease prompt to demolition and loss of the epithelial cells the respiratory mucosa. Influenza enters the host through the aviation routes. Influenza entanglements of the upper and lower respiratory tract are regular. These incorporate otitis media, sinusitis, bronchitis, and croup. Pneumonia is among the more serious difficulties of Influenza disease, an occasion most of the time seen in kids or grown-ups. Human Influenza prompts to complex cytopathic impacts because of down regulation of host cell protein combination and apoptosis, prevalently in the aviation routes epithelial cells. Apoptosis is interceded by both Fas-intervened systems and Fas-autonomous signs, which starts a caspase course. NA can actuate inactive TGF-β on the cell surface by encouraging cleavage of TGF-β into its dynamic shape that up-directs master apoptotic qualities. Apoptosis happens likewise in lymphocytes clarifying the lymphopenia seen amid intense disease.

 

  • Quantitative aspects of virus–cell interactions
  • Types of virus–host cell interactions
  • Cellular responses to viral infection
  • Host functions in viral replication and control
  • Replication, pathogenesis and transmission

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